This is the most effective way available for addressing nutritional deficiency associated with Parkinson's Disease.
89% of Parkinson’s disease patients take L-dopa (a dopamine precursor) with other medications.
Our approach does not use these traditional prescription drugs associated with Parkinson's disease care, which may cause:
- The 376% increase the Parkinson’s disease death rate.
- Known to cause and exacerbate nutritional deficiencies.
- Can induce nutritional deficiencies with symptoms identical to Parkinson’s disease symptoms.
- Can facilitate progression of brain damage causing Parkinson’s disease.
- Can cause the dopamine precursor to quit working.
- They are the primary cause of dyskinesias, and choreiform movement disorder thought to be due to dopamine precursors in the past.
Our Unique Approach
Permanent and irreversible brain damage is the cause of Parkinson’s disease. There is no cure. None. After the diagnosis of Parkinson’s disease occurs, it only gets worse.
A relative nutritional deficiency occurs when an optimal diet does not meet the needs of the system. In developing this approach we discovered that in the past doctors had wrongly attributed numerous relative nutritional deficiency symptoms to other causes. These relative nutritional deficiencies can have symptoms identical to Parkinson’s disease symptoms which are getting worse.
Inadequate systemic dopamine represents a relative nutritional deficiency of the nutrients the body uses to make dopamine. Normal people make enough dopamine from dietary nutrients. Parkinson’s disease patients do not make enough dopamine from an optimal diet. This dopamine deficiency represents a relative nutritional deficiency of the nutrients that the body requires to make dopamine.
The following is from the peer-reviewed scientific papers our doctors published:
- “A nutrient is any substance that facilitates normal system function. A drug is any substance that induces abnormal system function. A nutrient may become a drug. A drug may not become a nutrient. When a dopamine precursor is administered as a single agent, it may deplete serotonin, and would then be considered a drug, not a nutrient.” Neuropsychiatric Disease and Treatment 2016:12 763–775
When a dopamine precursor displays side effects it is no longer acting as a nutrient; it has become a drug. Dopamine precursors side effects represent imbalances between dopamine and other systems. The standard response of medicine to these nutritional deficiency driven symptoms is to give drugs, which do nothing to correct the nutritional deficiency and have the ability to make the nutritional deficiencies worse. A drug cannot effectively address a nutritional deficiency; only nutrients can.Improper management of the patient’s nutritional deficiencies leaves the patient with nutritional deficiency symptoms that have been confused with Parkinson’s disease symptoms, side effects, and deterioration in the past. So, what does the protocol do? Our approach is to put the patient in a state of normal feeling and normal function once again. First, we keep nutrients functioning as nutrients. Whenever nutrients in the form of dopamine precursors display side effects, they are functioning as a drug. Second, we optimally address the 29 relative nutritional deficiencies associated with Parkinson’s disease and its treatment. Only when we optimally address nutritional deficiencies can patients achieve optimal relief of symptoms with no side effects.